This study delves into the repercussions of the 2023 earthquake in Turkey, particularity its impact on air pollution. A year post-event, it is evident that scientific literature has paid limited attention to monitoring the situation. However, the release of hazardous substances, such as asbestos, lead, and other toxins, from damaged structures poses a significant threat by contaminating nearby air, soil, and water sources, thereby jeopardizing ecosystems and public well-being. The improper disposal of waste post-earthquake and the presence of mining and oil refinery sites in the region contribute to potential air pollutants. These circumstances create challenging environments conducive to the spread of respiratory diseases, with potential long-term health and social consequences. Unfortunately, existing data gaps hinder a comprehensive understanding of the situation. This paper pioneers the reporting and analysis of data regarding potential sources of air pollution resulting from the earthquake in Turkey. It also pinpoints gaps in knowledge, outlining areas that demand further investigation. To effectively prevent and mitigate air pollution risks and associated health concerns linked to earthquakes, strategic recommendations are proposed. A key suggestion is the establishment of post-disaster air pollution monitoring systems capable of swiftly identifying emerging health issues, facilitating efficient responses, and curtailing potential long-term effects of the disaster. The paper underscores the necessity for continuous health monitoring of the affected population to mitigate possible adverse impacts on human health. These strategies play a pivotal role in reducing the likelihood of air pollution, supporting emergency response and recovery initiatives, and fostering new dedicated scientific studies.

Microplastics' (MPs) ability to sorb and transport polychlorinated biphenyls (PCBs) in soil ecosystems warrants significant attention. Although organisms mainly encounter pollutants through the gut, the combined pollution impact of MPs and PCBs on soil fauna gut toxicity remains incompletely understood. Consequently, this study examined the gut toxicity of polystyrene MPs (PS-MPs) and PCB126 on Eisenia fetida, emphasizing the links between gut bacteria and bacterial translocation instigated by gut barrier impairment. Our findings underscored that E. fetida could ingest PS-MPs, which mitigated the PCB126 accumulation in E. fetida by 9.43 %. Exposure to PCB126 inhibited the expression of gut tight junction (TJ) protein genes. Although the presence of PS-MPs attenuated this suppression, it didn't alleviate gut barrier damage and bacterial translocation in the co-exposure group. This group demonstrated a significantly increased level of gut bacterial load (BLT, ANOVA, p = 0.005 vs control group) and lipopolysaccharide-binding protein (LBP, ANOVA, all p < 0.001 vs control, PCB, and PS groups), both of which displayed significant positive correlations with antibacterial defense. Furthermore, exposure to PS-MPs and PCB126, particularly within the co-exposure group, results in a marked decline in the dispersal ability of gut bacteria. This leads to dysbiosis (Adonis, R-2 = 0.294, p = 0.001), with remarkable signature taxa such as Janthinobacterium, Microbacterium and Pseudomonas, being implicated in gut barrier dysfunction. This research illuminates the mechanism of gut toxicity induced by PS-MPs and PCB126 combined pollution in earthworms, providing novel insights for the ecological risk assessment of soil.